Home MRI as Primary End Point for Pharmacologic Experiments of Liver Regeneration in a Murine Model of Partial Hepatectomy
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MRI as Primary End Point for Pharmacologic Experiments of Liver Regeneration in a Murine Model of Partial Hepatectomy

Rationale and Objectives

The study aimed to validate magnetic resonance imaging (MRI)-based liver volumetry as a quantitative measure of hepatic regeneration in mice subjected to partial hepatectomy, in view of routine in vivo pharmacologic studies characterizing compounds aiming to accelerate liver regeneration.

Materials and Methods

Partial hepatectomy was performed in male B6 mice ( n = 47). Images were acquired in 14.5 minutes from anesthetized and spontaneously respiring animals, without any gating and without administration of contrast material. Some of the mice ( n = 6) were treated with 1, 4-bis [2-(3, 5-dichloropyridyloxy)] benzene (TCPOBOP), a synthetic agonist of mouse constitutive androstane receptor, or with the corresponding vehicle ( n = 6). Postmortem analyses included total liver weight and histologic Ki67 expression.

Results

A highly significant correlation (R = 0.98, P = 1.5 × 10 −14 ) was obtained between the MRI-derived liver volumes and the postmortem liver weights in hepatectomized, untreated mice. MRI reliably monitored enhanced murine liver regrowth following treatment with TCPOBOP, as confirmed by comparative hepatocyte proliferation (Ki67 expression) and liver weight analysis (R = 0.96, P = 2 × 10 −6 ).

Conclusions

MRI-based monitoring of liver regrowth in mice without the requirement of euthanizing animals at several time points has been established. In comparison to terminal procedures, the number of hepatectomized mice needed to derive a liver (re)growth curve was reduced by a factor of 6. The feasibility of using this imaging approach in pharmacologic studies in the context of liver regeneration has been demonstrated.

Introduction

An efficient repair system of the liver tissue is required to ensure its proper functioning. The ability of the liver to regenerate itself as a response to loss of hepatic tissue has been known for a long time. This regenerative capacity may be impaired in cases of small-for-size liver grafts, liver resection (eg, tumor surgery), or chronic liver disease and acute liver failure often requiring liver transplant . Thus, there is a need for methods to enhance the intrinsic regeneration potential of the liver, enabling partial transplants, and to propagate hepatocytes ex vivo for use in cell transplant .

Whereas molecular mechanisms of liver cell growth and efficacy of mitogens can be studied in hepatic cell line-based in vitro systems, complex processes such as liver development or regeneration require in vivo models. The partial hepatectomy procedure in small rodents has constituted the most popular liver regeneration model. After surgical removal of three of the five liver lobes, the cells of the remaining two lobes proliferate until the liver regains its original size. Complete recovery takes approximately 8 days in rodents .

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Materials and Methods

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Animals

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Partial Hepatectomy

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TCPOBOP Treatment

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MRI

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MR Image Evaluation

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Liver Regeneration Index

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Liver regeneration index(%)=100×(Vt−VD1)/(Vbas−VD1) Liver regeneration index

(

%

)

=

100

×

(

V

t

V

D

1

)

/

(

V

b

a

s

V

D

1

)

with V t the liver volume at a given time point t, V D1 the liver volume on day 1 after partial hepatectomy, and V bas the liver volume at baseline prior to surgery. This index removes two possible sources of variance in the study, namely differences in initial liver volume and the volume of liver excised.

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Postmortem Analyses: Liver Weight and Histology

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Liver Function Tests

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Statistical Analyses

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Results

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Figure 1, Magnetic resonance images of the liver of spontaneously breathing mice. (a) Two representative coronal sections are shown, acquired from one mouse prior to and on days 1 and 8 after partial hepatectomy. The false color serves to illustrate the feasibility of segmenting the liver tissue for quantitation of the organ volume. (b) Evaluations of total liver volume made by two independent operators. (c) Corresponding Bland-Altman plot for the evaluations. The excellent agreement between both evaluators attests to the robustness of the approach. (Color version of figure is available online.)

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Figure 2, Liver regrowth curves obtained by magnetic resonance imaging. (a) Individual curves for n = 10 hepatectomized mice. (b) Mean liver volume (±SD, n = 10 mice). (c) Fitting of the mean curve showed that the regrowth process followed a sigmoidal pattern. (d) Regeneration curve (mean ± SD, n = 10 mice) derived from the liver volume. SD, standard deviation.

Figure 3, Comparison between the liver volume determined in vivo by magnetic resonance imaging (MRI) and the postmortem liver weight. Mice were euthanized immediately after the MRI acquisition on days 2 ( n = 8), 5 ( n = 7), and 9 ( n = 8) following hepatectomy.

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Figure 4, TCPOBOP treatment of hepatectomized mice. (a) Curve presenting the liver volume (means ± SD, n = 6 per group) after partial hepatectomy. TCPOBOP (1 mg/kg) was administered p.o. 2 hours before hepatectomy. The levels of significance **0.001 < P < 0.01 and *** P < 0.001 refer to comparisons to baseline volumes in each group. # P = 0.02, ## P = 0.003, and ### P < 0.001 are significant levels corresponding to comparisons between the groups at the given time points. (b) Regeneration curve (means ± SD) derived from the liver volume. ## P = 0.005 and ### P < 0.001 represent significant levels corresponding to comparisons between the groups at the given time points. (c) Liver weight determined immediately after harvesting relative to total body weight assessed immediately before necropsy (in percent) and Ki67 staining (means ± SD) demonstrating increased cell proliferation in the livers of TCPOBOP-treated mice at 24 hours after hepatectomy. For day 1 and day 2 posthepatectomy, n = 3 mice per group were sacrificed and the liver was analyzed postmortem. These mice were not imaged by MRI. For day 9, the livers of all mice measured by MRI ( n = 6 per group) were submitted to postmortem analyses. MRI, magnetic resonance imaging; SD, standard deviation.

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TABLE 1

Markers of Liver Function in the Blood Serum (Means ± Standard Deviation) on Day 9 After Hepatectomy

Parameter Vehicle TCPOBOP T-Prot (g/L) 55.4 ± 3.8 50.4 ± 2.3 (52.0–61.0) (48.0–54.0) Alb (g/L) 25.8 ± 1.5 22.6 ± 1.1 (24.0–28.0) (21.0–24.0) T-Bil (µmol/L) 8.8 ± 1.6 7.8 ± 4.1 (7.0–11.0) (5.0–15.0) AST (IU/L) 35.2 ± 5.9 47.0 ± 10.6 (29.0–45.0) (37.0–63.0) ALT (IU/L) 20.4 ± 3.0 60.0 ± 37.4 (17.0–24.0) (18.0–103.0)

Alb, albumin; ALT, alanine aminotransferase; AST, aspartate aminotransferase; T-Bil, total bilirubin; T-Prot, total protein.

The range of values obtained for a given parameter is provided in parentheses.

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Discussion and Conclusions

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Acknowledgments

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